Affordability of housing in the gap market: a case of Walmer link in Port Elizabeth, South Africa
- Makeleni, Nokukhanya Precious
- Authors: Makeleni, Nokukhanya Precious
- Date: 2019
- Subjects: Home ownership -- South Africa -- Port Elizabeth , Housing -- South Africa -- Port Elizabeth Public housing -- South Africa -- Port Elizabeth Rental housing
- Language: English
- Type: Thesis , Masters , MSc
- Identifier: http://hdl.handle.net/10948/42562 , vital:36668
- Description: The new dispensation, after 1994, in South Africa has been criticised a lot for not being able to satisfactorily achieve its election promise of redistribution and poverty alleviation. While success has been noted in the provision of low cost housing and subsequently home ownership for lower income earners, housing demand continues to be a challenge in the delivery capacity of housing for most South Africans. The people most excluded from homeownership include the public sector employees and laborers who face common, but different constraints. These people are classified as middle-income earners, they are either earn too much to qualify for a housing subsidy (RDP house), or too less to afford a house in the prime market. These people are referred to as the “gap market” because they fall with the gap of high and lowincome earners. Adopting a qualitative research method, survey questionnaire were sent to selective respondents involved in the development of affordable housing in the gap market in Walmer Link, Port Elizabeth, Nelson Mandela Bay Metropolitan area. This research sought to assess the affordability of housing in gap market, by understanding the challenges faced by the housing market in addressing the needs of the gap market in Walmer Link. Obstacles that hinders closure in the gap housing delivery, such as affordability, over indebtedness, poor credit ratings and inadequate supply for affordable housing in the gap market were identified during the research. While these obstacles show little indication of abating, this research’s findings and recommendations suggest new pathways for formulating new housing policies that address the housing backlog in the gap market. This also suggests that government policies are critical in developing a healthy and inclusive housing market.
- Full Text:
- Date Issued: 2019
- Authors: Makeleni, Nokukhanya Precious
- Date: 2019
- Subjects: Home ownership -- South Africa -- Port Elizabeth , Housing -- South Africa -- Port Elizabeth Public housing -- South Africa -- Port Elizabeth Rental housing
- Language: English
- Type: Thesis , Masters , MSc
- Identifier: http://hdl.handle.net/10948/42562 , vital:36668
- Description: The new dispensation, after 1994, in South Africa has been criticised a lot for not being able to satisfactorily achieve its election promise of redistribution and poverty alleviation. While success has been noted in the provision of low cost housing and subsequently home ownership for lower income earners, housing demand continues to be a challenge in the delivery capacity of housing for most South Africans. The people most excluded from homeownership include the public sector employees and laborers who face common, but different constraints. These people are classified as middle-income earners, they are either earn too much to qualify for a housing subsidy (RDP house), or too less to afford a house in the prime market. These people are referred to as the “gap market” because they fall with the gap of high and lowincome earners. Adopting a qualitative research method, survey questionnaire were sent to selective respondents involved in the development of affordable housing in the gap market in Walmer Link, Port Elizabeth, Nelson Mandela Bay Metropolitan area. This research sought to assess the affordability of housing in gap market, by understanding the challenges faced by the housing market in addressing the needs of the gap market in Walmer Link. Obstacles that hinders closure in the gap housing delivery, such as affordability, over indebtedness, poor credit ratings and inadequate supply for affordable housing in the gap market were identified during the research. While these obstacles show little indication of abating, this research’s findings and recommendations suggest new pathways for formulating new housing policies that address the housing backlog in the gap market. This also suggests that government policies are critical in developing a healthy and inclusive housing market.
- Full Text:
- Date Issued: 2019
Evaluation of model systems for the study of protein association / incorporation of Beta-Methylamino-L-Alanine (BMAA)
- Authors: Visser, Claire
- Date: 2011
- Subjects: Neurotoxic agents , Nervous system -- Diseases
- Language: English
- Type: Thesis , Masters , MSc
- Identifier: vital:10314 , http://hdl.handle.net/10948/1451 , Neurotoxic agents , Nervous system -- Diseases
- Description: β-methylamino-L-alanine (BMAA) is thought to be a contributing factor of Amyotrophic Lateral Sclerosis-Parkinsonism Dementia Complex (ALS/PDC). It has been shown that the levels of toxin ingestion by humans are too low to cause disease. However, it has recently been theorized that this toxin is bioaccumulated within cells. Via a process of slow release from this reservoir, the BMAA is able to bring about neurotoxicity. Mechanisms of uptake and bioaccumulation of BMAA have been proposed in several publications; however the mechanism of protein incorporation of BMAA has not yet been identified. Identifying suitable model systems would be a prerequisite in order for future studies on BMAA protein incorporation. Three specific models were therefore chosen for investigation; mammalian cell lines including C2C12 and HT29, a prokaryotic (E. coli) expression system and yeast cells. The cytotoxity of BMAA was established for the mammalian cell lines and further investigation of BMAA incorporation into cellular proteins was performed on all three above mentioned models. Samples were run on HPLC-MS in order to determine uptake of BMAA into cells or lack thereof. Results indicate negligible cytotoxicity as measured by MTT and CellTitre Blue assays, limited uptake and protein incorporation of BMAA within the prokaryotic model and insignificant uptake of BMAA by yeast cells. Although the uptake of BMAA in the prokaryotic model was not extensive, there was indeed uptake. BMAA was not only taken up into the cells but was also observed in inclusion body protein samples after hydrolysis. After further investigation and use, this model could very well provide researchers with information pertaining to the mechanism of association of BMAA with proteins. Although the other models provided negative results, this research was valuable in the sense that one can narrow down the number of possible model systems available. Also, in seeking models for studying protein association/incorporation, the use of the final target cell is not relevant or necessary as the purpose of the research was to identify a model system in which the mechanism of protein association/incorporation can, in future, be studied.
- Full Text:
- Date Issued: 2011
- Authors: Visser, Claire
- Date: 2011
- Subjects: Neurotoxic agents , Nervous system -- Diseases
- Language: English
- Type: Thesis , Masters , MSc
- Identifier: vital:10314 , http://hdl.handle.net/10948/1451 , Neurotoxic agents , Nervous system -- Diseases
- Description: β-methylamino-L-alanine (BMAA) is thought to be a contributing factor of Amyotrophic Lateral Sclerosis-Parkinsonism Dementia Complex (ALS/PDC). It has been shown that the levels of toxin ingestion by humans are too low to cause disease. However, it has recently been theorized that this toxin is bioaccumulated within cells. Via a process of slow release from this reservoir, the BMAA is able to bring about neurotoxicity. Mechanisms of uptake and bioaccumulation of BMAA have been proposed in several publications; however the mechanism of protein incorporation of BMAA has not yet been identified. Identifying suitable model systems would be a prerequisite in order for future studies on BMAA protein incorporation. Three specific models were therefore chosen for investigation; mammalian cell lines including C2C12 and HT29, a prokaryotic (E. coli) expression system and yeast cells. The cytotoxity of BMAA was established for the mammalian cell lines and further investigation of BMAA incorporation into cellular proteins was performed on all three above mentioned models. Samples were run on HPLC-MS in order to determine uptake of BMAA into cells or lack thereof. Results indicate negligible cytotoxicity as measured by MTT and CellTitre Blue assays, limited uptake and protein incorporation of BMAA within the prokaryotic model and insignificant uptake of BMAA by yeast cells. Although the uptake of BMAA in the prokaryotic model was not extensive, there was indeed uptake. BMAA was not only taken up into the cells but was also observed in inclusion body protein samples after hydrolysis. After further investigation and use, this model could very well provide researchers with information pertaining to the mechanism of association of BMAA with proteins. Although the other models provided negative results, this research was valuable in the sense that one can narrow down the number of possible model systems available. Also, in seeking models for studying protein association/incorporation, the use of the final target cell is not relevant or necessary as the purpose of the research was to identify a model system in which the mechanism of protein association/incorporation can, in future, be studied.
- Full Text:
- Date Issued: 2011
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